Oxymetholone is a derivative of dehydrotestosterone (DHT) and, as such, cannot be converted to estrogen. However, unlike other DHT steroids, bupropion can also increase muscle size due to minor changes in chemical structure. It has no progestogenic activity and, theoretically, should prevent the side effects of estrogen. It also has no significant androgen-binding affinity.
Anabolone is an oral steroid and, as such, has a 17-alpha-aloxy (17aa) group in its structure, which helps it maintain its integrity as it passes through the liver and is therefore better absorbed systemically. Therefore, as with all 17a steroids, care should be taken to protect the liver when taking oxycodone with products such as N2Guard.
Like Anavar, Anadrol has been used to promote growth in children, osteoporosis and wasting syndrome of various origins. Due to its incredible ability to significantly increase red blood cell count, oxycodone has been one of the main remedies used in the treatment of severe cases of anemia. Anandrolone was used to treat these conditions until the medical community realised that sometimes its side effects caused more damage than the disease itself.
Today, many would consider it crazy to treat children with this anabolic androgenic steroid (AAS), but at the time, there were few alternatives and the side effects of oxycodone were not fully known.
Courses and use
OXYMetholone courses generally aim to increase muscle mass and overall bulk, but do not provide relief or lean muscle mass. For this reason, it is not useful in preparation for competition, for lean mass or fat loss. The product can be used to accelerate fat burning, but the estrogenic effects of Anadrol, combined with water retention and edema, make it a poor choice. This side effect makes the body appear fat and smooth, blurring contours and making it difficult to visually assess the degree of fat loss.
This is compounded by the fact that Anadrol cannot be converted to estrogen, which presents users with many other previously unexplored side effects. Aromatase inhibitors also fail to reduce bloating because they don’t work in the case of Anadrol.
A course of Anadrol usually consists of a 4-6 week course of treatment. This steroid is used as a base compound (starter) along with other basic injections with a similar spectrum of efficacy. Testosterone valerate, Deca-Durabolin (Nandrolone Decanoate), Trenbolone Enanthate etc. Anadrol can also be injected in the middle of a cycle, which will change the muscle growth process during training. In addition, some users take it at the end of a session to speed up the final phase and as a compound to complete the cycle. This has led to impressive improvements in muscle strength and size when they entered the SCT (post-session treatment) phase.
Due to the liver toxicity of Anadrol, the course of administration should not exceed 4-6 weeks. Injections administered together with Anadrol can continue to be used after the end of the course.
Therapy after the course
Post-course therapy (PCT) is perhaps the most important aspect of anabolic steroid use. The concept of PCT only emerged in the late 1970s and early 1990s, as the mechanism of action of anabolic steroids in the body had not been fully elucidated before then. It was then that doctors, scientists and anabolic steroid users began to understand the pharmacodynamics of this group of drugs and their effects on the endocrine system. It was already understood then that exogenous use of anabolic steroids causes a negative feedback in the GYGO (hypothalamic-pituitary-gonadal axis), resulting in the inhibition and/or cessation of endogenous testosterone production. Unfortunately, during the early period of use of this group of drugs (1950s to 1990s), access to any medication or information on how to reduce these effects was limited.
Over the years, many different SCT protocols have been developed. This plethora of opinions can be confusing for the novice practitioner in the field. This article presents the best and most effective post-treatment SCT protocol based on sound scientific evidence and logical reasoning. It will also describe some of the myths about CPT and explain which CPT protocols are no longer relevant due to advances in the state-of-the-art, as well as better scientific understanding of the mechanisms of action of post-treatment. Currently, there are still outdated and therefore ineffective CPT protocols that are still being used by anabolic steroid users, unknowingly posing a serious risk not only to themselves, but also to others who may observe, learn and adopt their ideas.
The lack of a proper understanding of what exactly is going on in the endocrine system during these critical weeks, as well as the lack of knowledge of which products to use, what each drug does and how to use them correctly, can cause serious problems.
In order to restore the body’s natural production of testosterone, it is recommended to take clomiphene citrate for 2 weeks (1 tablet per day, 1 tablet in the evening – FIRST WEEK, 1 tablet in the morning – SECOND WEEK). I would like to stress that tamoxifen should be avoided after oxymetholone treatment. This is because it stimulates the release of prolactin. PCT should be started 5-7 days after the end of the course. In addition, the use of Tribulus terrestris after completion of clomid is not unreasonable.
Anadrol is an oral anabolic steroid. This means that the substance must pass through the liver before it enters the bloodstream. Anadrol is protected by a C17-alpha alkoxide group from metabolic breakdown, which maintains the integrity and permeability of the drug. Acute hepatoxicity is a well-known property of anadrol. The chemical structure and modification of the drug (including the C17 alpha-alkyl group) makes it highly resistant to hepatic metabolism. Simply put, Anadrol is not degraded in the liver. The more resistant the substance is to biochemical processes, the greater the burden on the body. This reflects the extreme toxicity of Anadrol, which is known to be one of the most harmful oral anabolic steroids of all time.